I noticed on time that i was was having this virus cos i immediately went to the hospital for a test when i started feeling some unusual pains and encountering some miserable symptoms in my body and i was diagnosed of Herpes simplex 2, i was so perplexed cos i heard there was no cure but i tried to still get rid of it and started to use really strong drugs from the hospital which really cost me a lot and all was to no avail.
I had to check on the internet one day bout this Herpes and how i could be helped and i keep seeing a lot of testimonies bout this man called Dr Mack, so i contacted him via his email and the mobile contact i saw there on the post of one Mrs Kathy Anne cos i needed this cure so bad as it has affected me deeply.
I contacted him and was optimistic that he would cure me, and he responded well which made me really calm, I did all that i was i was asked to do by him and which he sent me the herbal medicine with instructions to take it. Just in 14days i started to feel okay and noticed changes in me and later i went to do a test as he told me to, and which i was confirmed negative and Herpes free. Hi I have had herpes 2 for decades. Occurrences vary to 1 or 2 times a month too 3 or 4 times a year.
Mostly on my vagina but also on my buttocks. Doctor said I have a herpes and this is usually STI. Many years ago, I was working like a sailor on cargo ships. We got the huge forest trunks there. It was hot and humid. I realized on my lower back an insect and I inststinctavely killed it. It was a fly with yellow body striped like a Zebra.
Shortly I found on the a spot I killed the fly, painful and itchy pimples with yellow inside. Doctor said it probably Iprit Fly bite me and this insect leave eggs in human body. The live in Africa under trees core. If you think you might be at risk of developing shingles, a vaccine is available that might help protect you against shingles. Herpes simplex virus is transmitted through sexual activity and other skin-to-skin contact.
Having any type of sex without a condom or other barrier method puts you at risk of developing a herpes infection. Herpes simplex virus can be passed on even when a person is showing no symptoms at all. Unless you know your partner has tested negative for sexually transmitted infections STIs , always use protection. Protective barriers like condoms need to be used correctly to help prevent herpes simplex virus and other STIs from being passed on.
The virus can also be passed on no matter the gender of your sexual partner. You can receive treatment to manage your symptoms. For shingles, treatments help you during the 3 to 5 weeks your symptoms may last. For herpes, treatments help you address outbreaks as they happen.
Treatment can speed up the healing process and help you manage itching and pain, especially if you begin early after your symptoms appear. Testing may be free or offered at a low cost at a community clinic or your local health department. Shingles is generally diagnosed based on your symptoms. A medical professional will examine your rash and blisters and talk with you about your pain.
They might also send a small swab from one of your blisters for a lab test to determine if the varicella-zoster virus is present.
Herpes is diagnosed through a physical exam and lab tests. Just like with shingles, your doctor will examine your rash and take a swab from one of the blisters to send for a lab test. You might also have blood drawn to look for herpes simplex virus antibodies in your system.
Shingles and herpes are caused by two different viruses and are acquired in different ways. Shingles is treated with both antiviral prescription medications that can speed up your healing and with medications that can help with your pain. Your exact treatment plan will depend on how severe your case is, your overall health, and the medications you already take.
Antivirals can help you heal first and reduce your symptoms. Depending on your case and your overall health, you might take these medications during an outbreak or daily.
Talk to a doctor about the use of any of the treatments above. These treatments should not be used without guidance by a medical professional so that you can help avoid any unintended side effects, such as allergic reactions.
Each condition has its own telltale signs. Most patients have been infected by varicella prior to HIV. In the rare occurrence of varicella in an HIV infected person, IV acyclovir treatment should be considered as visceral dissemination is not uncommon. Susceptible patients without history of chickenpox and antibody should be given varicella zoster immune globulin VZIG as prophylaxis within 96 hours of exposure. Note that VZIG is ineffective as treatment.
As a live attenuated vaccine, the varicella vaccine is contraindicated in HIV patients. It is important to advise patient on the use of barrier contraceptives and the knowledge that shedding of virus is often subclinical. Sex should be abstained when there are recognisable recurrences. The use of condom reduces but does not eliminate the risk of infection as some genital lesions may not be covered by the condom. Both acyclovir and valacyclovir can be considered.
Breakthrough recurrences do not necessarily reflect drug resistance, and higher doses of suppressive therapy may be attempted. However, this approach may involve a long period of antiviral treatment and the decision needs to be individualised.
The frequency of recurrent genital herpes diminishes over time in many patients, and the patient's psychological adjustment to the disease might change. Therefore, periodically during suppressive treatment e. An alternative approach is to treat each recurrent episode to ameliorate the symptoms. It has also been shown that while HAART does reduce the incidence of symptomatic genital herpes and its severity, it has a modest and not statistically significant effect on subclinical HSV-2 reactivation.
Genital herpes in pregnancy is an important issue. However, caesarean section does not completely eliminate the risk of HSV transmission to the infant. The safety of systemic acyclovir, valacyclovir, and famciclovir therapy in pregnant women has not been definitively established. However, available data do not indicate an increased risk for major birth defects in women treated with these antiviral agents during the first trimester.
Acyclovir may be administered orally to pregnant women with first episode genital herpes or severe recurrent herpes and should be administered IV to pregnant women with severe HSV infection. Acyclovir treatment late in pregnancy reduces the frequency of caesarean sections among women who have recurrent genital herpes by diminishing the frequency of recurrences at term, and many specialists recommend such treatment.
Increase in rates of herpes simplex virus type 1 as a cause of anogenital herpes in western Sydney, Australia, between and Sex Transm Infect ; Hong Kong Department of Health.
Epidemiology of herpes simplex virus type 2 in Hong Kong. Difference in seroprevalence of herpes simplex virus type 2 infection among antenatal women in Hong Kong and southern China.
Oxford: Oxford University Press, ; p Herpes simplex virus 2 infection increases HIV acquisition in men and women: systematic review and meta-analysis of longitudinal studies. AIDS ; The effects of herpes simplex virus-2 on HIV-1 acquisition and transmission: a review of two overlapping epidemics. J Acquir Immune Defic Syndr ; Herpes simplex virus infection induces replication of human immunodeficiency virus type 1.
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J Infect Dis ; JAMA ; Increased genital shedding of herpes simplex virus type 2 in HIV-seropositive women. Ann Intern Med ; Disseminated herpes zoster in patients with human immunodeficiency virus infection.
Am J Med ; Herpes zoster: a possible early clinical sign for development of acquired immunodeficiency syndrome in high-risk individuals. J Am Acad Dermatol ; Varicella-zoster virus retinitis in a patient with AIDS-related complex: case report and brief review of the acute retinal necrosis syndrome. Clin Infect Dis ; Triggers for reactivation of latent disease include stress, fever, immunocompromised state, damage to local tissue, and ultraviolet light.
Risk factors for acquiring genital disease are age 15 to 30 years, increased number of sexual partners, black or Hispanic race, and HIV positivity. Varicella zoster virus infection: Individual lesions of varicella zoster may look exactly like herpes simplex, with clustered vesicles or ulcers on an erythematous base. Varicella zoster tends to follow a dermatomal distribution, which can help to distinguish from herpes simplex.
Disseminated herpes simplex and disseminated zoster may be indistinguishable clinically. Aphthous ulcers: These occur most commonly in the mouth but can also involve the genitals, such as in Behcet disease. Large aphthous ulcers can be associated with HIV infection. These most commonly occur on the mucosal inner lips, tongue, floor of the mouth, and inner cheeks. They occur as small round ulcers with a yellow or grey ulcer floor, which often occur singly or in a linear fashion.
They usually heal within 1 week. HIV infection: HIV may present with major aphthous ulcerations, which occur most commonly on the oral mucosa. Serologic tests can show primary seroconversion for HSV-1 or HSV-2 infection; however, it does not definitively diagnose active disease. Tzank smear: Scraping of the base of an early unroofed blister can demonstrate virally infected multinucleated epithelial giant cells. Viral tissue culture: This may be positive within 48 hours and can allow for resistance testing if needed.
HSV deoxyribonucleic acid detection: Gene amplification by PCR, ligase chain reaction, or other methods can be done on skin lesions or cerebral spinal fluid when evaluating for encephalitis and other infected tissue. Direct fluorescent antibody: Cells scraped from the base of an early unroofed blister are stained with a direct fluorescent antibody. Imaging studies are only useful when there is suspected HSV encephalitis. Brain imaging studies, such as computed tomography and magnetic resonance imaging scans, can be performed to look for involvement of the temporal lobe.
If you decide the patient has herpes simplex virus infection, what therapies should you initiate immediately? Dermatology would be most helpful in diagnosing this infection when there is skin or mucous membrane involvement. If the patients are immunocompetent, no therapy may be necessary since the lesions usually self-resolve. If the patient is immunocompromised, severely symptomatic, or disseminated or the lesions are extensive, treatment is needed.
Recommended medications for initial or recurrent infection include aciclovir, valaciclovir, and famciclovir all evidence category A. Aciclovir resistant infection can be treated with intravenous foscarnet or topical cidofovir evidence category C. Complications of severe oral herpes include dysphagia, severe pain, and inability to take oral medications.
In HIV infection, oral or genital herpes can be persistent and cause deep painful ulcers. Bacterial and yeast superinfections can occur in patients with persistent ulcerations. Ocular infection can occur, particularly in association with oral herpes infection. Complications of genital herpes include dysuria, pain, and edema.
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